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PLoS Med 5:e185

9 , 10 ENDS use was also implicated in adverse effects on the head, neck, oral cells regarding aberrant morphology, cytotoxicity, reduced viability, delayed fibroblast migration and genotoxicity

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reported the administration of peg-ADA to reduce extracellular adenosine levels in an LPS-induced mouse model of lung injury compromised resolution of pulmonary inflammation ( Purinergic Signaling in Chronic Pulmonary Inflammation ATP, adenosine, CD73, and CD39 have also been implicated in the pathogenesis of chronic lung inflammation (Figure 4)