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Study 1 assessed the acute effects of Cagrilintide in 32 twelve-week-old male WT mice fed a standard chow diet of 3.14 kcal/g

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GLP-1 receptor activation can reduce neuroinflammation and oxidative stress, and restore insulin signaling, which may compensate for the dysfunctional dopaminergic neurotransmission and motor deficits associated with PD pathogenesis [74, 75]

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