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Several studies have identified various sources of ROS and RNS in IBD, including neutrophils, macrophages, and inflamed intestinal tissue ( 2 Mechanism of oxidative stress in IBD While the precise mechanisms responsible for the development of IBD remain unclear, it is widely accepted that multiple factors contribute to its etiology, including overproduction of ROS, damage to biomolecules, mitochondrial dysfunction, recruitment of immune cells, impaired antioxidant defense system, and the activation of inflammatory pathways ( Figure 1 )
Its design supports multiple withdrawals from each vial, providing flexibility and efficiency for subcutaneous or intramuscular administration
L-ergothioneine modulates oxidative damage in the kidney and liver of rats in vivo : studies upon the profile of polyunsaturated fatty acids
