slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROS–cytochrome c–caspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROS–cytochrome c–caspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model

Name: slc25a39 is necessary for mitochondrial glutathione import in mammalian cells identified as a key regulator of hepatocellular carcinoma progression through the ROS–cytochrome c–caspase signaling axis | Cellular & Molecular Biology Letters Development of a mouse model
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Dal Pan 2009)

For instance, female mice administered BSO exhibited much higher susceptibility to acetaminophen-induced hepatotoxicity than male mice [43], while male mice treated with BSO were much more susceptible to thiabendazole nephrotoxicity than females [44]