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Indeed, cAMP levels can be elicited by GLP-1 treatment of mouse cardiomyocytes without increasing intracellular Ca 2+ or inducing contractility [38]

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42,43 In addition, the prevalence of AMS is obviously higher in subjects with patent foramen ovale (PFO), an embryologic residual right-to-left cardiac shunt that can impair pulmonary gas exchange efficiency, than without PFO