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The bridge from genes changed to cell function changed is still half-built, and most of the published work leans on dermal fibroblasts, with far less on the cardiac, lung, or joint varieties, which makes generalizing across tissue types shakier than it sounds

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Unlike the lymphocytic inflammation seen in SLE-specific lesions, the inflammation in bullous LE is neutrophilic, the blister is subepidermal, and an antibody against type VII collagen is seen in the blood