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AOD-9604 Modified fragment of human growth hormone (hGH 176-191) that has been studied for its ability to stimulate fat metabolism and promote weight reduction without increasing IGF-1 activity

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GNE inhibition results in hyposialylation of IGF-1R Since the key function of GNE is epimerization and phosphorylation of precursors in the sialic acid biosynthetic pathway, absence of functional GNE would cause hyposialylation and affect sialylation of cell surface receptors 15,30