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(Poster presentation) Campbell, J

Luteolin-3-O-Phosphate Inhibits Lipopolysaccharide-Induced Inflammatory Responses by Regulating NF-B/MAPK Cascade Signaling in RAW 264.7 Cells

Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia

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