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Immune Modulation at the Maternal-Fetal Interface Research has demonstrated that hCG modulates immune responses at the maternal-fetal interface through epigenetic mechanisms specifically, hCG inhibits CXCL10 expression in human endometrial stromal cells by enhancing EZH2 expression and inducing H3K27me3 histone methylation at the CXCL10 promoter, thereby suppressing T cell recruitment to the implantation site
Research has demonstrated that GHK-type peptides can downregulate transforming growth factor-beta 1 (TGF-1), a cytokine implicated in premature follicle regression and the shift from anagen to catagen
Compounds sold as research peptides are intended for laboratory research only and are not substitutes for FDA-approved medications or clinician-supervised care
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