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The reasons underlying insufficient efficacy of vessel-targeting strategies have been extensively investigated, and include stroma and tumor cell mechanisms of resistance [287, 297]

Research focus centers on sustained receptor engagement and how the compound facilitates communication across expansive metabolic frameworks

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Experimental data indicate that GHK-Cu can influence collagen and glycosaminoglycan synthesis, modulate proteoglycan and chondroitin sulfate turnover, and recruit fibroblasts, immune cells, and endothelial cells to sites of tissue injury