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These cell lines have defects affecting the enzymes involved in the respiratory chain and are, therefore, unable to generate an electrochemical gradient across the mitochondrial membrane, concomitant with the loss of the glucose-induced increase of intracellular Ca 2+ [1113]

Findings from an in vivo animal-based experiment suggest that the biologically active compound gypenoside (GP-14) mitigates neuroinflammation and BBB disruption in a mouse model of HACE