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Description

Implication of trimethylamine N-oxide (TMAO) in disease: potential biomarker or new therapeutic target

Sony Corporation, Tokyo, Japan)

They enhance insulin secretion in response to meals, suppress glucagon release, slow gastric emptying, and reduce appetite through central nervous system pathways

We have shown the generality of the combinatorial ubiquitin code with other TRIP12 substrates, suggesting that certain OTUD5-protected substrates are also regulated by DUB-resistant K29-linked ubiquitin chains