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CPNE1 achieves this by upregulating markers such as CD163, CD206, and interleukin-10 (IL-10), which are characteristic of M2 macrophages

By reprogramming these M2-like TAMs into the pro-inflammatory, tumor-inhibiting M1 phenotype, researchers aim to enhance anti-tumor immune responses and overcome resistance to therapies such as immune checkpoint blockade (ICB) and CAR-T cell therapy

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