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PNPLA3 can remodel the phospholipid monolayer in LDs (79) by transferring polyunsaturated FAs from TAG to phospholipids via transacylation, or possibly through TAG hydrolase activity (80) and can promote FA re-esterification during lipolysis possibly through lysophosphatidic acid acyltransferase (LPAAT) or transacylase activity (70)

Sections were deparaffinized in a xylene ethanol series and placed in Tris-EDTA (TE) buffer for antigen retrieval (10 mM Tris, 1 mM EDTA, 0.05% Tween, pH = 9.0) and then blocked in 5% BSA

17851792, Lee, Chia-Lun, et al

In contrast, in experimental autoimmune encephalomyelitis (EAE) models, exendin-4, liraglutide, and dulaglutide consistently attenuate inflammation and oxidative stress (81, 87, 182), reduce clinical severity, and improve motor outcomes (83), mediated by AMPK/SIRT1 activation and NLRP3 inhibition