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Collectively, these mitochondrial dysfunction-associated events contribute to ferroptosis-related nucleus pulposus cell injury and extracellular matrix degradation in IDD, thereby suggesting that targeting mitochondrial dysfunction may represent a potential therapeutic strategy for IDD

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In developing countries, individuals with unrepaired clefts cannot eat or speak properly, are kept from attending school or obtaining employment and may be subjected to further abuse and cruelty as a result of their birth defect