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Alteration of gene expression by chromosome loss in the postnatal mouse brain

Due to prolonged exposure to oxidative stress during the prenatal period, it is postulated that a decline in the compensatory antioxidant capacity of cells may exacerbate oxidative-related comorbidities, including cognitive phenotypes

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The accumulation of these cells can induce a local or systemic inflammatory state, subsequently causing bone marrow suppression and heart dysfunction [60]