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An analysis of N-acetylcysteine treatment for acetaminophen overdose using a systems model of drug-induced liver injury

Multiple pathways of NAD + synthesis have been identified, which are differentially active in tissues and utilize various molecular precursors to ensure maintenance of NAD + homeostasis and counteract NAD + turnover

Subsequent work reported that T4 promotes oligodendrogenesis in the demyelinating CNS and drives OPC differentiation, with p38 MAPK signaling and myelin-basic-protein synthesis implicated in the pathway