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found that MAM-resident protein FUNDC2 participated in ferroptosis by regulating mitochondrial GSH levels through the FUNDC2-SLC25A11 axis in doxorubicin-induced cardiomyopathy[60]

The observed upregulation of not only the ThDP-dependent dehydrogenases limiting and feeding the TCA cycle, but also the thiamine transporters and ThDP-producing enzyme TPK1 (Figures 4B, 5B), may compensate for decreased levels and/or increased demands of thiamine in enterocytes 69 months after gastric bypass, facilitated by the higher expression of the proteins binding thiamine or ThDP

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