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The disruption extends beyond direct receptor binding-EED exposure significantly impairs recruitment of critical coactivators such as steroid receptor coactivator-1 (SRC-1), substantially weakening AR transcriptional capacity (35)

The ways to inhibit GPX4 directly mainly include covalently binding GPX4 and suppressing its expression

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ThDP is used as a cofactor for the microbial enzyme pyruvate-ferredoxin oxidoreductase, which catalyzes the conversion of pyruvate to acetyl-CoA in the pathway for butyrate synthesis (56)