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Dysbiosis in inflammatory bowel diseases: the oxygen hypothesis
69 In gastric cancer cells, the absence of CDO1 contributed to restore cellular glutathione levels, to prevent ROS and lipid peroxidation, and to promote resistance to ferroptosis, a type of programmed cell death dependent on iron and associated with lipid peroxide accumulation
Dietary restriction of cysteine and methionine sensitizes gliomas to ferroptosis and induces alterations in energetic metabolism
Shortly after these studies began, it was proclaimed in the medical community that glutathione is our body's most powerful antioxidant and the main detoxifying agent in the body, protecting cells from free radicals and oxidative stress and boosting the immune system
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