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ROS acts as a central hub connecting ferroptosis to PCa pathogenesis, amplified by inflammatory signals and redox-sensitive transcription factors (e.g., Nrf2, HIF-1) that either directly enhance oxidative stress or indirectly modulate ferroptosis susceptibility [Figure 2A]

Fibre adds bulk to stools and promotes regular bowel movements, but increasing too quickly can worsen bloating, especially when taking a GLP-1 receptor agonist

Numerous studies have identified many target genes of NRF2 and revealed functions of pleiotropic NRF2 in addition to its redox-regulating function

G., Vernacotola, S., Peruzzi, L., Tabolacci, E., Mila, M., Militerni, R., Musumeci, S., Ramos, F