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Ferroptosis in Epilepsy Previous studies have shown that early persistent dysregulation of iron metabolism and activation of antioxidant signals are pathological features of focal cortical dysplasia type IIb (FCD IIb) and tuberous sclerosis complex (TSC) (Zimmer et al., 2020)

In this context, PH20, I-BET151, and PXS-5153A may indirectly counteract thyroid-related fibrosis by degrading ECM, inhibiting profibrotic signaling, or reducing collagen crosslinking

The latter effect is the result of the reduced ability for platelets to adhere to exposed sub-endothelial extracellular matrix collagen which is required for their activation

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