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Additionally, noise exposure and cochlear ablation upregulate BDNF expression in the inferior colliculus alongside GAD65 (glutamate decarboxylase), suggesting that BDNF participates in the excitatory/inhibitory balance within central auditory structures following acoustic insult (Erol et al., 2012)

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[80] found that selenium could inhibit ferroptosis through the Nrf2/GPX4 signaling pathway and ameliorate autistic-like behaviors in mice

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