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This explains why high levels of oxidative stress is implicated not just in neurodegenerative conditions but cardiovascular diseases as well

Takizawa T, Huang IY, Ikuta T, Yoshida A

This reprogramming likely serves two primary functions: (1) redirecting saturated fatty acids away from membrane lipid synthesis to mitigate lipotoxic stress, particularly as stearyl desaturase 1 (SCD1) and fatty acid oxidation (FAO) are inhibited due to limited O availability, and (2) increasing lipid storage capacity to buffer against energy fluctuations [7173]

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