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The chronic TNFR1 activation by the soluble form of TNF- triggers amyloid genesis process by enhancing the protein expression and activity of -secretase through the NF-B pathway, resulting in increased A production and plaque formation [85]

In: Cochrane Handbook for Systematic Reviews of Interventions New Jersey: Wiley-Blackwell

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Although fibrosis improvement remains limited, they manage metabolic liver conditions by reducing inflammation and fat deposition, and combining them with anti-inflammatory therapies may enhance the therapeutic efficacy (Table 1 shows representative clinical trials and results of GLP-1RAs in liver diseases