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It is important to note that claims about increased energy expenditure in humans remain insufficiently evidenced at this stage, and the net glycaemic effects reflect observed trial outcomes rather than a straightforward mechanistic reduction in hepatic glucose output the concurrent glucagon receptor agonism may, in fact, influence hepatic glucose production in a complex and not fully characterised manner

Strong healthcare infrastructure, established reimbursement systems, and widespread physician adoption further support market penetration

doi: 10.3390/antiox9050375 215

Cellular energy therapies may benefit individuals experiencing fatigue, metabolic decline, or reduced physical performance