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As a result, the regulatory framework governing compounded GLP-1 medications continues to change

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In hepatocytes, FXR activation induces the transcription of the SHP, which suppresses cholesterol 7-hydroxylase (CYP7A1), the rate-limiting enzyme in BA synthesis ( de novo fatty acid synthesis ( Based on the immunomodulatory mechanisms of FXR, therapeutic strategies targeting FXR have shown promise for treating related disorders, including nonalcoholic steatohepatitis (NASH), primary biliary cholangitis (PBC), primary sclerosing cholangitis (PSC), and type 2 diabetes (44)