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343 Similar to observations in AD, the reprogramming of microglial energy metabolism is implicated in the pathogenesis of PD

Research has characterised its capacity to modulate hundreds of genes associated with tissue remodelling, antioxidant defence and extracellular matrix synthesis, with established roles in collagen and glycosaminoglycan production

Glucocorticoid impaired the wound healing ability of endothelial progenitor cells by reducing the expression of CXCR4 in the PGE2 pathway

It was engineered to keep GH's effect on fat metabolism while leaving out the parts that raise blood sugar, raise IGF-1, or trigger growth