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Adverse events collected were: angina (change in frequency), stroke, transient ischemic attack, MI, congestive heart failure, cardiac revascularization, cerebral revascularization, peripheral revascularization, high blood pressure and diabetes (new diagnosis only)

Thioredoxin 1 is inactivated due to oxidation induced by peroxiredoxin under oxidative stress and reactivated by the glutaredoxin system

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Since NRF2 activation induces the antioxidant and detoxifying response to restore redox homeostasis state, in recent years there has been a growing interest in identifying and evaluating new compounds capable of targeting the KEAP1-NRF2 system, to induce cytoprotective response against OS