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At the signaling level, the cascade initiated by ipamorelin binding to GHS-R1a involves phospholipase C activation and inositol trisphosphate (IP3) generation, leading to calcium release from intracellular stores a pathway distinct from the GHRH receptor, which primarily signals through cyclic AMP

[191] highlighted the potential benefits of a multimodal strategy that combines pharmaceutical, physical, and ALA therapy for the treatment of sciatic neuropathy in real-world settings

Acknowledgements This work was supported by the Israel Science Foundation founded by the Israel Academy of Sciences and Humanities, grant number 419/15 and by the Gurwin Fund for Scientific Research