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The published literature is the answer to whether the molecule lives up to its reputation
Finally, unbiased phosphoproteomics combined with kinase inhibition has revealed hundreds of novel GSK3 substrates [34], many of which have known or plausible roles in GLUT4 traffic including the regulator of GLUT4 endocytosis GAPVD1/GAPEX5 (GSK3 sites: S758 and T762 in mice) [106108], the early/recycling endosome regulator RBSN/Rabenosyn-5 (S225) [109], and the endosome-to-Golgi trafficking regulator GOLGA4/golgin-245 (T39 and S40) [110]
If you want a more general overview of symptom patterns, our GLP-1 side effects guide is a useful reference
Jain, A., et al.,p62/SQSTM1 is a target gene for transcription factor NRF2 and creates a positive feedback loop by inducing antioxidant response element-driven gene transcription
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