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It was previously reported that manganese may protect from cadmium toxicity in cultured cells and reduce cellular cadmium accumulation, presumably because of competition for uptake via SLC39A14 (Himeno et al, 2009)

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Extended ATX supplementation, spanning a period of 8 months, led to a partial reversal of Erastin-induced alterations in human LECs from ARC models, which included the suppression of GPX4, an accumulation of ROS, and a decline in mitochondrial membrane potential (144)