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Accordingly, Masson staining revealed that recombinant CXCL10 treatment sharply increased fibrosis in the CXCL10 group, whereas IL18R1 antibody treatment reversed fibrosis to levels close to those of the control in the 10+18ab group (Fig

doi: 10.1016/j.ekir.2022.12.014 130 DhillonS

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