semaglutide cardiac effects ameliorates pressure overload-induced hypertrophy by improving cardiac mitophagy to suppress the activation of NLRP3 inflammasome Semaglutide and Cardiovascular Outcomes in

The primary endpoint was total fluid intake during the evaluation visits

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1,2 These activities are constantly challenged by intrinsic and extrinsic perturbations, including increased protein synthesis, ER-Ca 2+ depletion, dysregulated redox homeostasis, energy deprivation, hypoglycemia, hypoxia, and inflammatory stimuli, resulting in improper protein conformation and misfolding, often referred to as nonnative or aberrant conformation

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