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Abstract Renal fibrosis represents the final common pathological pathway of nearly all chronic kidney disease (CKD), yet effective therapeutic options remain profoundly limited

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Furthermore, microbial-derived lipopolysaccharides (LPS) released during dysbiosis can breach a compromised gut barrier and trigger Toll-like receptor 4 (TLR4) signaling in Kupffer cells, leading to cytokine release, hepatic lipid deposition, and fibrosis

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