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97 Hyperuricemia, by inducing oxidative stress and inflammation, diminishes the expression of eNOS and the synthesis of NO, while elevating levels of inflammatory cytokines such as IL-6 and TNF- , ultimately impairing endothelial function

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The continuous progression of the above pathological processes ultimately leads to renal tubular atrophy, renal interstitial fibrosis, and progressive irreversible decline of renal function, which is the core pathological mechanism of cisplatin-induced AKI to CKD progression (Fig.2) (Li et al

Intravenous administration must be performed slowly (23 minutes)