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Animal studies have shown that TMAO induces vascular inflammation by activating the sirtuin 3 -superoxide dismutase 2- mitochondrial ROS (SIRT3SOD2mtROS) and stimulating in vitro and in vivo formation of the NLRP3 inflammasome (Seldin et al., 2016)

The effects persisted beyond the peptide's pharmacokinetic half-life, supporting the "trigger" hypothesis (PMID 514474)

Possible mechanisms of action of LC Direct effects LC has been reported to maintain cellular energy [34], reduce oxidative stress [72] and minimize cell death by apoptosis [33], which are necessary for proper oocyte growth and maturation of blastocyst

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