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In OA research, recent evidence indicates that pharmacological activation of the Nrf2GPX4 signaling axis using bioactive natural products or synthetic small molecule agonists can effectively mitigate ferroptosis in vitro cultured chondrocytes and in vivo OA animal models, while significantly suppressing ECM degradation progression (Zhang Z

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Generally, low antibody titers are detected in a minority of patients, and higher titers develop in a much smaller proportion of patients (4952)